- In CARTITUDE-4, CARVYKTI® demonstrated clinically meaningful
improvements in patient-reported outcomes when compared to standard
of care
- The as-treated population in CARTITUDE-4 demonstrated strong
rates of progression-free survival and overall response
- Longer-term data from CARTITUDE-2 showed deep and durable
responses in earlier lines of treatment among patients in Cohort A
and Cohort B
Legend Biotech Corporation (NASDAQ: LEGN) (Legend Biotech), a
global biotechnology company developing, manufacturing, and
commercializing novel therapies to treat life-threatening diseases,
announced today patient-reported outcome (PRO) data from the Phase
3 CARTITUDE-4 study from an oral presentation at the 2023 American
Society of Hematology (ASH) Annual Meeting (Abstract #1063). These
data showed clinically meaningful improvement in health-related
quality of life following a single CARVYKTI® (ciltacabtagene
autoleucel; cilta-cel) infusion in adults lenalidomide-refractory
multiple myeloma (MM) who received one to three prior lines of
therapy (LOT), compared to patients treated with the standard of
care (SOC) treatment regimens of either pomalidomide, bortezomib
and dexamethasone (PVd) or daratumumab, pomalidomide and
dexamethasone (DPd).1 The PRO data also demonstrated meaningful
reductions in disease-specific symptoms after a single infusion for
patients in the CARVYKTI® arm, while patients in the SOC treatment
arm trended toward worsening or lower degrees of improvement from
baseline for most domains and symptoms.
Eligible patients in the CARTITUDE-4 study had
lenalidomide-refractory MM, and had one to three prior LOT,
including a proteasome inhibitor (PI) and an immunomodulatory drug.
Four hundred nineteen patients were randomized, with 208 patients
in the CARVYKTI® arm and 211 patients in the SOC arm. At the
clinical cut-off on November 1, 2022, 99 patients in the CARVYKTI®
arm and 66 patients in the SOC arm had baseline and 12-month PRO
assessments, representing data prior to disease progression. When
compared to SOC, patients who received the CARVYKTI® infusion
exceeded clinically meaningful thresholds for average improvement
from baseline to 12 months in global health status (10.1 points vs.
-1.5 points), pain (-10.2 points vs. -3.9 points), and the visual
analogue scale (8.0 points vs. 1.4 points).1
“The CARTITUDE-4 data presented today reinforce the impact that
a single infusion of CARVYKTI® may provide to patients,” said
Roberto Mina, Assistant Professor, Division of Hematology,
Department of Molecular Biotechnology and Health Sciences,
University of Torino, Turin, Italy.
When compared to SOC, the PRO data for CARVYKTI® neared
clinically meaningful thresholds when evaluating improvements in
fatigue (-9.1 points vs. 2.8 points) and emotional functioning (9.5
points vs. 2.2 points), and numerically favored CARVYKTI® for all
other domains established by the European Organisation for Research
and Treatment of Cancer Quality of Life Questionnaire Core 30
(EORTC QLQ-C30; 100-point scale). The median time until MM symptom
worsening in the CARVYKTI® arm was 23.7 months compared to 18.9
months in the SOC arm (hazard ratio [HR], 0.42), as measured with
the Multiple Myeloma Symptom and Impact Questionnaire (MySIm-Q;
5-point scale).1
CARTITUDE-4 As-Treated Analysis
Illustrated Favorable Progression-Free Survival (PFS)
Rate
An additional analysis of the CARTITUDE-4 study data was
presented as a poster (Abstract #4866) at the ASH Annual Meeting.
At the clinical cut-off, 176 of the 208 patients were randomized to
the CARVYKTI® treatment arm. The median age of this patient
population was 61 years and 34 percent had received 1 prior LOT. At
a median follow-up of 16 months following randomization, 22 percent
of patients received one bridging therapy cycle, 59 percent
received two cycles and 18 percent received 3 cycles, and disease
burden was effectively controlled across the as-treated patient set
during bridging therapy.2
At 12 months following infusion, the PFS rate was 85 percent,
and the overall survival (OS) rate was 92 percent. Median PFS had
not been reached. The overall response rate (ORR) was 99 percent
and 86 percent of patients achieved complete response or better
(>CR). Of the minimum residual
disease- (MRD) evaluable patients (n=144), 77 percent achieved both
MRD negativity and >CR.2
The most common CAR-T cell-related toxicity was Cytokine Release
Syndrome (CRS) at 76 percent (1 percent grade 3), the neurotoxicity
rate was 21 percent (3 percent grade 3/4), and Immune Effector
Cell-Associated Neurotoxicity Syndrome (ICANS) occurred in 5
percent of patients (no grade 3/4). Other neurotoxicities occurred
in 17 percent of patients (2 percent grade 3/4). By the clinical
cut-off, CRS and ICANS had resolved in all patients.2
Data from CARTITUDE-2 Cohorts A and B
Demonstrated Deep and Durable Responses
During a second oral presentation, longer term efficacy and
safety data from CARTITUDE-2 cohorts A and B were also presented at
the ASH Annual Meeting (Abstract #1021). At a median follow-up of
approximately 29 months, patients with lenalidomide-refractory MM
after one to three lines of therapy (Cohort A) and those with early
relapse (Cohort B) that were treated with CARVYKTI® in earlier
lines of therapy experienced deep and durable responses .3
In both Cohort A (n=20) and Cohort B (n=19), treatment with
CARVYKTI® led to overall response rates of 95 percent (≥CR, 90
percent) and 100 percent (≥CR, 90 percent), respectively. In Cohort
A, the 24-month PFS rate was 75 percent, and the 24-month OS rate
was 75 percent. As for cohort B, the 24-month PFS and OS rates were
73 percent and 84 percent, respectively. There were no new
CAR-T-related safety signals for Cohorts A and B, however one
additional CAR-T related cell neurotoxicity (grade 2) was reported
in cohort B.3
“We believe the safety and efficacy data presented from the
CARTITUDE Clinical Development program at the 2023 ASH Annual
Meeting support our continuous efforts to bring CARVYKTI® to
myeloma patients in various stages of disease progression,” said
Ying Huang, Ph.D., Chief Executive Officer of Legend Biotech. “Part
of our mission is to improve the lives of patients worldwide, and
we are excited that the CARTITUDE-4 PRO analyses indicate that
patients may experience a higher health-related quality of life
following a single CARVYKTI® infusion.”
Disclosure: Dr. Mina has provided consulting, advisory, and
speaking services to Legend Biotech
CARVYKTI® Important Safety Information
CARVYKTI® INDICATIONS AND USAGE
CARVYKTI® (ciltacabtagene autoleucel) is a B-cell maturation
antigen (BCMA)-directed genetically modified autologous T cell
immunotherapy indicated for the treatment of adult patients with
relapsed or refractory multiple myeloma, after four or more prior
lines of therapy, including a proteasome inhibitor, an
immunomodulatory agent, and an anti-CD38 monoclonal antibody.
CARVYKTI® IMPORTANT SAFETY INFORMATION
WARNING: CYTOKINE RELEASE SYNDROME, NEUROLOGIC TOXICITIES,
HLH/MAS, and PROLONGED and RECURRENT CYTOPENIA
Cytokine Release Syndrome (CRS), including fatal or
life-threatening reactions, occurred in patients following
treatment with CARVYKTI®. Do not administer CARVYKTI® to patients
with active infection or inflammatory disorders. Treat severe or
life-threatening CRS with tocilizumab or tocilizumab and
corticosteroids.
Immune Effector Cell-Associated Neurotoxicity Syndrome (ICANS),
which may be fatal or life-threatening, occurred following
treatment with CARVYKTI®, including before CRS onset, concurrently
with CRS, after CRS resolution, or in the absence of CRS. Monitor
for neurologic events after treatment with CARVYKTI®. Provide
supportive care and/or corticosteroids as needed.
Parkinsonism and Guillain-Barré syndrome and their associated
complications resulting in fatal or life-threatening reactions have
occurred following treatment with CARVYKTI®.
Hemophagocytic Lymphohistiocytosis/Macrophage Activation
Syndrome (HLH/MAS), including fatal and life-threatening reactions,
occurred in patients following treatment with CARVYKTI®. HLH/MAS
can occur with CRS or neurologic toxicities.
Prolonged and/or recurrent cytopenias with bleeding and
infection and requirement for stem cell transplantation for
hematopoietic recovery occurred following treatment with
CARVYKTI®.
CARVYKTI® is available only through a restricted program under a
Risk Evaluation and Mitigation Strategy (REMS) called the CARVYKTI®
REMS Program.
WARNINGS AND PRECAUTIONS
CYTOKINE RELEASE SYNDROME (CRS) including fatal or
life-threatening reactions, occurred following treatment with
CARVYKTI® in 95% (92/97) of patients receiving ciltacabtagene
autoleucel. Grade 3 or higher CRS (2019 ASTCT grade) occurred in 5%
(5/97) of patients, with Grade 5 CRS reported in 1 patient. The
median time to onset of CRS was 7 days (range: 112 days). The most
common manifestations of CRS included pyrexia (100%), hypotension
(43%), increased aspartate aminotransferase (AST) (22%), chills
(15%), increased alanine aminotransferase (ALT) (14%) and sinus
tachycardia (11%). Grade 3 or higher events associated with CRS
included increased AST and ALT, hyperbilirubinemia, hypotension,
pyrexia, hypoxia, respiratory failure, acute kidney injury,
disseminated intravascular coagulation, HLH/MAS, angina pectoris,
supraventricular and ventricular tachycardia, malaise, myalgias,
increased C-reactive protein, ferritin, blood alkaline phosphatase
and gamma-glutamyl transferase.
Identify CRS based on clinical presentation. Evaluate for and
treat other causes of fever, hypoxia, and hypotension. CRS has been
reported to be associated with findings of HLH/MAS, and the
physiology of the syndromes may overlap. HLH/MAS is a potentially
life-threatening condition. In patients with progressive symptoms
of CRS or refractory CRS despite treatment, evaluate for evidence
of HLH/MAS.
Sixty-nine of 97 (71%) patients received tocilizumab and/or a
corticosteroid for CRS after infusion of ciltacabtagene autoleucel.
Forty-four (45%) patients received only tocilizumab, of whom 33
(34%) received a single dose and 11 (11%) received more than one
dose; 24 patients (25%) received tocilizumab and a corticosteroid,
and one patient (1%) received only corticosteroids. Ensure that a
minimum of two doses of tocilizumab are available prior to infusion
of CARVYKTI®.
Monitor patients at least daily for 10 days following CARVYKTI®
infusion at a REMS-certified healthcare facility for signs and
symptoms of CRS. Monitor patients for signs or symptoms of CRS for
at least 4 weeks after infusion. At the first sign of CRS,
immediately institute treatment with supportive care, tocilizumab,
or tocilizumab and corticosteroids.
Counsel patients to seek immediate medical attention should
signs or symptoms of CRS occur at any time.
NEUROLOGIC TOXICITIES, which may be severe,
life-threatening or fatal, occurred following treatment with
CARVYKTI®. Neurologic toxicities included ICANS, neurologic
toxicity with signs and symptoms of parkinsonism, Guillain-Barré
Syndrome, peripheral neuropathies, and cranial nerve palsies.
Counsel patients on the signs and symptoms of these neurologic
toxicities, and on the delayed nature of onset of some of these
toxicities. Instruct patients to seek immediate medical attention
for further assessment and management if signs or symptoms of any
of these neurologic toxicities occur at any time.
Overall, one or more subtypes of neurologic toxicity described
below occurred following ciltacabtagene autoleucel in 26% (25/97)
of patients, of which 11% (11/97) of patients experienced Grade 3
or higher events. These subtypes of neurologic toxicities were also
observed in two ongoing studies.
Immune Effector Cell-Associated Neurotoxicity Syndrome (ICANS):
Patients may experience fatal or life-threatening ICANS following
treatment with CARVYKTI®, including before CRS onset, concurrently
with CRS, after CRS resolution, or in the absence of CRS. ICANS
occurred in 23% (22/97) of patients receiving ciltacabtagene
autoleucel including Grade 3 or 4 events in 3% (3/97) and Grade 5
(fatal) events in 2% (2/97). The median time to onset of ICANS was
8 days (range 1-28 days). All 22 patients with ICANS had CRS. The
most frequent (≥5%) manifestation of ICANS included encephalopathy
(23%), aphasia (8%) and headache (6%).
Monitor patients at least daily for 10 days following CARVYKTI®
infusion at the REMS-certified healthcare facility for signs and
symptoms of ICANS. Rule out other causes of ICANS symptoms. Monitor
patients for signs or symptoms of ICANS for at least 4 weeks after
infusion and treat promptly. Neurologic toxicity should be managed
with supportive care and/or corticosteroids as needed.
Parkinsonism: Of the 25 patients in the CARTITUDE-1 study
experiencing any neurotoxicity, five male patients had neurologic
toxicity with several signs and symptoms of parkinsonism, distinct
from immune effector cell-associated neurotoxicity syndrome
(ICANS). Neurologic toxicity with parkinsonism has been reported in
other ongoing trials of ciltacabtagene autoleucel. Patients had
parkinsonian and non-parkinsonian symptoms that included tremor,
bradykinesia, involuntary movements, stereotypy, loss of
spontaneous movements, masked facies, apathy, flat affect, fatigue,
rigidity, psychomotor retardation, micrographia, dysgraphia,
apraxia, lethargy, confusion, somnolence, loss of consciousness,
delayed reflexes, hyperreflexia, memory loss, difficulty
swallowing, bowel incontinence, falls, stooped posture, shuffling
gait, muscle weakness and wasting, motor dysfunction, motor and
sensory loss, akinetic mutism, and frontal lobe release signs. The
median onset of parkinsonism in the 5 patients in CARTITUDE-1 was
43 days (range 15-108) from infusion of ciltacabtagene
autoleucel.
Monitor patients for signs and symptoms of parkinsonism that may
be delayed in onset and managed with supportive care measures.
There is limited efficacy information with medications used for the
treatment of Parkinson’s disease, for the improvement or resolution
of parkinsonism symptoms following CARVYKTI® treatment.
Guillain-Barré Syndrome: A fatal outcome following
Guillain-Barré Syndrome (GBS) has occurred in another ongoing study
of ciltacabtagene autoleucel despite treatment with intravenous
immunoglobulins. Symptoms reported include those consistent with
Miller-Fisher variant of GBS, encephalopathy, motor weakness,
speech disturbances and polyradiculoneuritis.
Monitor for GBS. Evaluate patients presenting with peripheral
neuropathy for GBS. Consider treatment of GBS with supportive care
measures and in conjunction with immunoglobulins and plasma
exchange, depending on severity of GBS.
Peripheral Neuropathy: Six patients in CARTITUDE-1 developed
peripheral neuropathy. These neuropathies presented as sensory,
motor, or sensorimotor neuropathies. Median time of onset of
symptoms was 62 days (range 4-136 days), median duration of
peripheral neuropathies was 256 days (range 2-465 days) including
those with ongoing neuropathy. Patients who experienced peripheral
neuropathy also experienced cranial nerve palsies or GBS in other
ongoing trials of ciltacabtagene autoleucel.
Cranial Nerve Palsies: Three patients (3.1%) experienced cranial
nerve palsies in CARTITUDE1. All three patients had 7th cranial
nerve palsy; one patient had 5th cranial nerve palsy as well.
Median time to onset was 26 days (range 21-101 days) following
infusion of ciltacabtagene autoleucel. Occurrence of 3rd and 6th
cranial nerve palsy, bilateral 7th cranial nerve palsy, worsening
of cranial nerve palsy after improvement, and occurrence of
peripheral neuropathy in patients with cranial nerve palsy have
also been reported in ongoing trials of ciltacabtagene autoleucel.
Monitor patients for signs and symptoms of cranial nerve palsies.
Consider management with systemic corticosteroids, depending on the
severity and progression of signs and symptoms.
HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS (HLH)/MACROPHAGE
ACTIVATION SYNDROME (MAS): Fatal HLH occurred in one patient
(1%), 99 days after ciltacabtagene autoleucel. The HLH event was
preceded by prolonged CRS lasting 97 days. The manifestations of
HLH/MAS include hypotension, hypoxia with diffuse alveolar damage,
coagulopathy, cytopenia, and multi-organ dysfunction, including
renal dysfunction. HLH is a life-threatening condition with a high
mortality rate if not recognized and treated early. Treatment of
HLH/MAS should be administered per institutional standards.
CARVYKTI® REMS: Because of the risk of CRS and neurologic
toxicities, CARVYKTI® is available only through a restricted
program under a Risk Evaluation and Mitigation Strategy (REMS)
called the CARVYKTI® REMS.
Further information is available at
https://www.carvyktirems.com/ or 1-844-672-0067.
PROLONGED AND RECURRENT CYTOPENIAS: Patients may exhibit
prolonged and recurrent cytopenias following lymphodepleting
chemotherapy and CARVYKTI® infusion. One patient underwent
autologous stem cell therapy for hematopoietic reconstitution due
to prolonged thrombocytopenia.
In CARTITUDE-1, 30% (29/97) of patients experienced prolonged
Grade 3 or 4 neutropenia and 41% (40/97) of patients experienced
prolonged Grade 3 or 4 thrombocytopenia that had not resolved by
Day 30 following ciltacabtagene autoleucel infusion.
Recurrent Grade 3 or 4 neutropenia, thrombocytopenia,
lymphopenia, and anemia were seen in 63% (61/97), 18% (17/97), 60%
(58/97), and 37% (36/97) after recovery from initial Grade 3 or 4
cytopenia following infusion. After Day 60 following ciltacabtagene
autoleucel infusion, 31%, 12% and 6% of patients had a recurrence
of Grade 3 or higher lymphopenia, neutropenia and thrombocytopenia,
respectively, after initial recovery of their Grade 3 or 4
cytopenia. Eighty-seven percent (84/97) of patients had one, two,
or three or more recurrences of Grade 3 or 4 cytopenias after
initial recovery of Grade 3 or 4 cytopenia. Six and 11 patients had
Grade 3 or 4 neutropenia and thrombocytopenia, respectively, at the
time of death.
Monitor blood counts prior to and after CARVYKTI® infusion.
Manage cytopenias with growth factors and blood product transfusion
support according to local institutional guidelines.
INFECTIONS: CARVYKTI® should not be administered to
patients with active infection or inflammatory disorders. Severe,
life-threatening or fatal infections occurred in patients after
CARVYKTI® infusion.
Infections (all grades) occurred in 57 (59%) patients. Grade 3
or 4 infections occurred in 23% (22/97) of patients; Grade 3 or 4
infections with an unspecified pathogen occurred in 17%, viral
infections in 7%, bacterial infections in 1%, and fungal infections
in 1% of patients. Overall, four patients had Grade 5 infections:
lung abscess (n=1), sepsis (n=2) and pneumonia (n=1).
Monitor patients for signs and symptoms of infection before and
after CARVYKTI® infusion and treat patients appropriately.
Administer prophylactic, pre-emptive and/or therapeutic
antimicrobials according to the standard institutional guidelines.
Febrile neutropenia was observed in 10% of patients after
ciltacabtagene autoleucel infusion and may be concurrent with CRS.
In the event of febrile neutropenia, evaluate for infection and
manage with broad-spectrum antibiotics, fluids, and other
supportive care, as medically indicated.
Viral Reactivation: Hepatitis B virus (HBV) reactivation, in
some cases resulting in fulminant hepatitis, hepatic failure and
death, can occur in patients with hypogammaglobulinemia. Perform
screening for Cytomegalovirus (CMV), HBV, hepatitis C virus (HCV),
and human immunodeficiency virus (HIV), or any other infectious
agents if clinically indicated in accordance with clinical
guidelines before collection of cells for manufacturing. Consider
antiviral therapy to prevent viral reactivation per local
institutional guidelines/clinical practice.
HYPOGAMMAGLOBULINEMIA was reported as an adverse event in
12% (12/97) of patients; laboratory IgG levels fell below 500 mg/dL
after infusion in 92% (89/97) of patients. Monitor immunoglobulin
levels after treatment with CARVYKTI® and administer IVIG for IgG
<400 mg/dL. Manage per local institutional guidelines, including
infection precautions and antibiotic or antiviral prophylaxis.
Use of Live Vaccines: The safety of immunization with live viral
vaccines during or following CARVYKTI® treatment has not been
studied. Vaccination with live virus vaccines is not recommended
for at least 6 weeks prior to the start of lymphodepleting
chemotherapy, during CARVYKTI® treatment, and until immune recovery
following treatment with CARVYKTI®.
HYPERSENSITIVITY REACTIONS have occurred in 5% (5/97) of
patients following ciltacabtagene autoleucel infusion. Serious
hypersensitivity reactions, including anaphylaxis, may be due to
the dimethyl sulfoxide (DMSO) in CARVYKTI®. Patients should be
carefully monitored for 2 hours after infusion for signs and
symptoms of severe reaction. Treat promptly and manage
appropriately according to the severity of the hypersensitivity
reaction.
SECONDARY MALIGNANCIES: Patients may develop secondary
malignancies. Monitor life-long for secondary malignancies. In the
event that a secondary malignancy occurs, contact Janssen Biotech,
Inc., at 1-800-526-7736 for reporting and to obtain instructions on
collection of patient samples for testing of secondary malignancy
of T cell origin.
EFFECTS ON ABILITY TO DRIVE AND USE MACHINES: Due to the
potential for neurologic events, including altered mental status,
seizures, neurocognitive decline, or neuropathy, patients are at
risk for altered or decreased consciousness or coordination in the
8 weeks following CARVYKTI® infusion. Advise patients to refrain
from driving and engaging in hazardous occupations or activities,
such as operating heavy or potentially dangerous machinery during
this initial period, and in the event of new onset of any
neurologic toxicities.
ADVERSE REACTIONS
The most common non-laboratory adverse reactions (incidence
greater than 20%) are pyrexia, cytokine release syndrome,
hypogammaglobulinemia, hypotension, musculoskeletal pain, fatigue,
infections of unspecified pathogen, cough, chills, diarrhea,
nausea, encephalopathy, decreased appetite, upper respiratory tract
infection, headache, tachycardia, dizziness, dyspnea, edema, viral
infections, coagulopathy, constipation, and vomiting. The most
common laboratory adverse reactions (incidence greater than or
equal to 50%) include thrombocytopenia, neutropenia, anemia,
aminotransferase elevation, and hypoalbuminemia.
Please read full Prescribing Information including Boxed Warning
for CARVYKTI®.
ABOUT CARVYKTI® (CILTACABTAGENE AUTOLEUCEL;
CILTA-CEL)
Ciltacabtagene autoleucel is a B-cell maturation antigen
(BCMA)-directed, genetically modified autologous T-cell
immunotherapy, which involves reprogramming a patient’s own T-cells
with a transgene encoding a chimeric antigen receptor (CAR) that
identifies and eliminates cells that express BCMA. BCMA is
primarily expressed on the surface of malignant multiple myeloma
B-lineage cells, as well as late-stage B-cells and plasma cells.
The cilta-cel CAR protein features two BCMA-targeting single domain
antibodies designed to confer high avidity against human BCMA. Upon
binding to BCMA-expressing cells, the CAR promotes T-cell
activation, expansion, and elimination of target cells.4
In December 2017, Legend Biotech entered into an exclusive
worldwide license and collaboration agreement with Janssen Biotech,
Inc. (Janssen) to develop and commercialize cilta-cel.
ABOUT CARTITUDE-4
CARTITUDE-4 (NCT04181827) is an ongoing, international,
randomized, open-label Phase 3 study evaluating the efficacy and
safety of cilta-cel versus pomalidomide, bortezomib and
dexamethasone (PVd) or daratumumab, pomalidomide and dexamethasone
(DPd) in adult patients with relapsed and lenalidomide-refractory
multiple myeloma who received one to three prior lines of therapy,
including a PI and an IMiD. The primary endpoint of the study was
progression-free survival.5
ABOUT CARTITUDE-2
CARTITUDE-2 (NCT04133636) is an ongoing Phase 2 multicohort
study evaluating the safety and efficacy of cilta-cel in various
clinical settings (Cohorts A, B, C, D, E, F, G, H). The primary
study objective is to measure the percentage of patients with
negative minimal residual disease (MRD).6
ABOUT MULTIPLE MYELOMA
Multiple myeloma is an incurable blood cancer that starts in the
bone marrow and is characterized by an excessive proliferation of
plasma cells.7 In 2023, it is estimated that more than 35,000
people will be diagnosed with multiple myeloma, and more than
12,000 people will die from the disease in the U.S.8 While some
patients with multiple myeloma initially have no symptoms, most
patients are diagnosed due to symptoms that can include bone
problems, low blood counts, calcium elevation, kidney problems or
infections.9
ABOUT LEGEND BIOTECH
Legend Biotech is a global biotechnology company dedicated to
treating, and one day curing, life-threatening diseases.
Headquartered in Somerset, New Jersey, we are developing advanced
cell therapies across a diverse array of technology platforms,
including autologous and allogeneic chimeric antigen receptor
T-cell, gamma-delta T cell (γδ T) and natural killer (NK)
cell-based immunotherapy. From our three R&D sites around the
world, we apply these innovative technologies to pursue the
discovery of safe, efficacious and cutting-edge therapeutics for
patients worldwide.
Learn more at www.legendbiotech.com and follow us on X (formerly
Twitter) and LinkedIn.
CAUTIONARY NOTE REGARDING FORWARD-LOOKING STATEMENTS
Statements in this press release about future expectations,
plans and prospects, as well as any other statements regarding
matters that are not historical facts, constitute “forward-looking
statements” within the meaning of The Private Securities Litigation
Reform Act of 1995. These statements include, but are not limited
to, statements relating to Legend Biotech’s expectations for
cilta-cel, expectations for Legend Biotech’s product candidates
based on clinical trial results,
the potential effect of treatment with cilta-cel and the
potential benefits of Legend Biotech’s product candidates. The
words “anticipate,” “believe,” “continue,” “could,” “estimate,”
“expect,” “intend,” “may,” “plan,” “potential,” “predict,”
“project,” “should,” “target,” “will,” “would” and similar
expressions are intended to identify forward-looking statements,
although not all forward-looking statements contain these
identifying words. Actual results may differ materially from those
indicated by such forward-looking statements as a result of various
important factors. Legend Biotech’s expectations could be affected
by, among other things, uncertainties involved in the development
of new pharmaceutical products; unexpected clinical trial results,
including as a result of additional analysis of existing clinical
data or unexpected new clinical data; unexpected regulatory actions
or delays, including requests for additional safety and/or efficacy
data or analysis of data, or government regulation generally;
unexpected delays as a result of actions undertaken, or failures to
act, by our third party partners; uncertainties arising from
challenges to Legend Biotech’s patent or other proprietary
intellectual property protection, including the uncertainties
involved in the U.S. litigation process; competition in general;
government, industry, and general product pricing and other
political pressures; the duration and severity of the COVID-19
pandemic and governmental and regulatory measures implemented in
response to the evolving situation; as well as the other factors
discussed in the “Risk Factors” section of Legend Biotech’s Annual
Report on Form 20-F filed with the Securities and Exchange
Commission on March 30, 2023 and other filings and furnishings made
by Legend Biotech with the U.S. Securities and Exchange Commission
on EDGAR at www.sec.gov. Should one or more of these risks or
uncertainties materialize, or should underlying assumptions prove
incorrect, actual results may vary materially from those described
in this press release as anticipated, believed, estimated or
expected. Any forward-looking statements contained in this press
release speak only as of the date of this press release. Legend
Biotech specifically disclaims any obligation to update any
forward-looking statement, whether as a result of new information,
future events or otherwise.
References
1 Mina, R. Patient-Reported Outcomes in the Phase 3 CARTITUDE-4
Study of Ciltacabtagene Autoleucel Vs Standard of Care in Patients
with Lenalidomide-Refractory Multiple Myeloma after 1-3 Lines of
Therapy. Abstract #1063 [Oral Presentation]. Presented at the 2023
American Society of Hematology Annual Meeting.
2 Sidiqi, M. H. Efficacy and Safety in Patients with
Lenalidomide-Refractory Multiple Myeloma after 1-3 Prior Lines Who
Received a Single Infusion of Ciltacabtagene Autoleucel As Study
Treatment in the Phase 3 CARTITUDE-4 Trial. Abstract #4866 [Poster
Presentation]. Presented at the 2023 American Society of Hematology
Annual Meeting.
3 Hillengass, J. The Phase 2 CARTITUDE-2 Trial: Updated Efficacy
and Safety of Ciltacabtagene Autoleucel in Patients with Multiple
Myeloma and 1–3 Prior Lines of Therapy (Cohort A) and with Early
Relapse after First Line Treatment (Cohort B) Abstract #1021 [Oral
Presentation]. Presented at the 2023 American Society of Hematology
Annual Meeting.
4 CARVYKTI Prescribing Information. Horsham, PA: Janssen
Biotech, Inc.
5 ClinicalTrials.gov. A Study Comparing JNJ-68284528, a CAR-T
Therapy Directed Against B-cell Maturation Antigen (BCMA), Versus
Pomalidomide, Bortezomib and Dexamethasone (PVd) or Daratumumab,
Pomalidomide and Dexamethasone (DPd) in Participants With Relapsed
and Lenalidomide-Refractory Multiple Myeloma (CARTITUDE-4).
Available at: https://clinicaltrials.gov/study/NCT04181827. Last
accessed Nov 2023.
6 ClinicalTrials.gov. A Study of JNJ-68284528, a Chimeric
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View source
version on businesswire.com: https://www.businesswire.com/news/home/20231211889964/en/
Press: Alexandra Ventura, Corporate Communications &
Investor Relations, Legend Biotech alex.ventura@legendbiotech.com
732-850-5598
Investor: Jessie Yeung, Head of Investor Relations & Public
Relations, Legend Biotech jessie.yeung@legendbiotech.com
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